| Type | Description |
|---|---|
| Definition | CCR4-NOT transcription complex, subunit 3 |
| Date | Results | Publications |
|---|---|---|
| 2020-01-04 12:17:00 | Cnot3 plays a crucial role in liver functional maturation | 30733279 |
| 2019-09-21 11:01:00 | Data indicate that autophagy-related protein 7 (Atg7) promotes cardiac dysfunction of CCR4-NOT complex component Cnot3 knockout (Cnot3 mKO) mice independently of the canonical autophagy pathway. | 29438013 |
| 2017-11-26 00:29:00 | we propose that CNOT3 maintains the pluripotent state by promoting differentiation gene mRNA deadenylation and degradation, and we identify poly(A) tail-length regulation as a post-transcriptional mechanism that controls pluripotency. | 27746116 |
| 2017-05-13 16:28:00 | Cnot3(ad-/-) mice exhibit lipodystrophy. | 28032897 |
| 2017-05-13 12:09:00 | increases the number of ALP-positive colonies after iPSC induction and decreases expression levels of Eomes and p21 mRNAs. Based on these observations, we propose that the CCR4-NOT deadenylase activity contributes to iPSC induction. | 27037025 |
| Type | IDs |
|---|---|
| Synonymous | A930039N10Rik |
| Gene |
UniProtKB-ID:
CNOT3_MOUSE
UniprotKB:
Q8K0V4
UniParc:
UPI0000028292
EMBL:
BC030332,
BC141041,
BC053437
Ensembl:
ENSMUSG00000035632
KO:
mmu:232791
|
| Nucleutide sequences |
EMBL-CDS:
AAI41042.1,
AAH30332.1,
AAH53437.1
Ensembl_TRS:
ENSMUST00000038913
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000039098
RefSeq:
XP_036008814.1,
XP_036008813.1,
XP_030098269.1,
XP_006539819.1,
XP_036008820.1,
XP_036008817.1,
XP_030098267.1,
XP_036008822.1,
XP_017177635.1,
XP_030098266.1,
XP_006539816.1,
XP_036008812.1,
XP_036008824.1,
XP_030098268.1,
XP_036008816.1,
XP_036008815.1,
XP_006539818.1,
XP_036008818.1,
NP_666288.1,
XP_036008823.1,
XP_036008819.1,
XP_006539815.1,
XP_036008821.1
|
| Others |
UniRef100:
UniRef100_Q8K0V4
UniRef90:
UniRef90_Q8K0V4
UniRef50:
UniRef50_Q8K0V4
UniGene:
Mm.358731
CCDS:
CCDS20721.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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