Type | Description |
---|---|
Definition | serum amyloid A 2 |
Date | Results | Publications |
---|---|---|
2016-04-23 11:40:00 | SAA1/2 produced by macrophages promotes early lesion formation in the ascending aorta in LDLR knockout mice. | 26187995 |
2014-10-18 12:49:00 | CE/J mice possess functional Saa1 and Saa2 genes with identical amino acid sequence. | 24228751 |
2014-09-27 10:14:00 | Its gene hold broader diversity and greater complexity and these characteristics were likely attained through gene duplication and repeated gene conversion events in the Mus lineage. | 24521869 |
2014-03-22 11:21:00 | The absence of endogenous SAA1.1 and 2.1 does not affect atherosclerotic lipid deposition in apolipoprotein E-deficient mice fed either normal or Western diets. | 24265416 |
2013-12-07 11:23:00 | High level SAA expression induced amyloidosis in all mice after a short, slightly variable delay. | 23959890 |
Type | IDs |
---|---|
Synonymous | AW111173, Saa-2, Saa1 |
Gene |
UniProtKB-ID:
SAA2_MOUSE
UniprotKB:
P05367
UniParc:
UPI0000020FCE
EMBL:
M17791,
M11130,
M13522
Ensembl:
ENSMUSG00000057465
KO:
mmu:20209
|
Nucleutide sequences |
EMBL-CDS:
AAA40085.1,
AAA40086.1,
AAA40087.1
Ensembl_TRS:
ENSMUST00000075982,
ENSMUST00000210769
|
Protein sequencees |
Ensembl_PRO:
ENSMUSP00000075365,
ENSMUSP00000147751
RefSeq:
NP_035444.1,
NP_001366197.1,
NP_001366198.1,
NP_001344420.1
|
Others |
UniRef100:
UniRef100_P05367
UniRef90:
UniRef90_P05367
UniRef50:
UniRef50_P05367
UniGene:
Mm.200941
CCDS:
CCDS21285.1
|
{{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
---|---|---|---|---|---|---|---|---|
Refseq |
{{protein.nucleotideVersion}}
Ensembl: {{protein.nucleotideEnsembl}} |
{{protein.proteinVersion}}
Ensembl: {{protein.proteinEnsembl}} |
{{uniprot}} , |
Definition: {{{protein.definition}}}Transcript Veriant:{{protein.transcriptVeriant}} Status: {{protein.status}} |
||||
Location | {{protein.contigId}} ( {{protein.positionStart}}..{{protein.positionEnd}} , {{protein.orientation}} ) | |||||||
Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
{{conservedDomain.comments == '' || conservedDomain.comments == null ? "-" : conservedDomain.comments }} |
Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
{{conservedDomain.comments == '' || conservedDomain.comments == null ? "-" : conservedDomain.comments }} |
暂无数据
排名 | 科研单位 | 文献 |
---|---|---|
{{affIndex+1}} |
{{aff.value}} |
{{aff.size}} |
目前还没有研究热点单位
排名 | 研究人员 | 文献 |
---|---|---|
{{authorIndex+1}} |
{{author.value}} |
{{author.size}} |
目前还没有研究热点人员