| Type | Description |
|---|---|
| Definition | NePHronoPhthisis (human kidney disease) homolog |
| Date | Results | Publications |
|---|---|---|
| 2016-07-30 11:05:00 | OSM-3 mutations cause NPHP-4 dependent and independent phenotypes. | 26863025 |
| 2016-07-28 13:16:00 | NPHP-1 and NPHP-4 proteins play important and redundant roles in facilitating ciliary sensory signal transduction. | 15817158 |
| 2010-06-28 12:11:00 | data indicate a genetic interaction between mutations in mks-3 and nphp-4 causes cilia and sensilla morphology defects | 20150540 |
| 2010-01-21 00:00:00 | The C. elegans B9 proteins form a complex that localizes to the base of cilia. Mutations in the B9 genes do not overtly affect cilia formation unless they are in combination with a mutation in nph-1 or nph-4. | 18337471 |
| 2010-01-21 00:00:00 | The NPHP homologs may be necessary for initial assembly of the cilium, whereas the polycystic kidney disease homologs may function as sensory transducers. | 15659564 |
| Type | IDs |
|---|---|
| Gene |
UniProtKB-ID:
G5ECP0_CAEEL
UniprotKB:
G5ECP0
UniParc:
UPI00004F8D02
EMBL:
BX284605,
AY959881
EnsemblGenome:
WBGene00011261
KO:
cel:CELE_R13H4.1
|
| Nucleutide sequences |
EMBL-CDS:
CAB04654.3,
AAX56914.1
Gene_ORFName:
CELE_R13H4.1,
R13H4.1
|
| Protein sequencees |
RefSeq:
NP_505982.2
|
| Others |
UniRef100:
UniRef100_G5ECP0
UniRef90:
UniRef90_G5ECP0
UniRef50:
UniRef50_G5ECP0
UniGene:
Cel.34216
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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