LOV1 - NB-ARC domain-containing disease resistance protein-BMKCloudï½ç¾è¿å®¢äº

例如："NBL1", "4681", "drought"

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837635 LOV1

基因描述

Type	Description
Definition	NB-ARC domain-containing disease resistance protein

研究结论

Date	Results	Publications
2013-09-07 11:45:00	These results provide evidence that the victorin response mediated by LOV1 is a defense response.	23634836
2012-11-24 10:38:00	in LOV1's absence, victorin inhibits TRX-h5, resulting in compromised defense but not disease by C. victoriae; in LOV1's presence, victorin binding to TRX-h5 activates LOV1 and elicits a resistance-like response that confers disease susceptibility	23087001
2010-05-03 11:19:00	LONG VEGETATIVE PHASE 1 controls flowering time and cold response in Arabidopsis	17653269
2010-03-08 12:04:00	Photochemical properties of phot1 rely on interactions between LOV1 and LOV2. LOV1 cannot operate as a dark-state repressor, resulting in constitutive receptor autophosphorylation and accelerated internalization from the plasma membrane.	19880798
2010-01-21 00:00:00	LOV1 may function as a resistance gene to a naturally-occurring pathogen of Arabidopsis.[LOV1]	18052878

名称对应

Type	IDs
Synonymous	LOCUS ORCHESTRATING VICTORIN EFFECTS1, T19D16.16, T19D16_16
Gene	UniProtKB-ID: LOV1A_ARATH, A0A1P8AT99_ARATH UniprotKB: O04093, A0A1P8AT99 UniParc: UPI00084882F7, UPI0000196C25 EMBL: CP002684, AY074545, U95973 EnsemblGenome: AT1G10920 KO: ath:AT1G10920
Nucleutide sequences	EMBL-CDS: AAB65485.1, AEE28663.1, ANM59880.1, AEE28664.1, ANM59881.1, ANM59882.1 Gene_ORFName: T19D16.16, T19D16.16, T19D16_16 EnsemblGenome_TRS: AT1G10920.2, AT1G10920.1, AT1G10920.4
Protein sequencees	EnsemblGenome_PRO: AT1G10920.2, AT1G10920.1, AT1G10920.4 RefSeq: NP_172561.2, NP_001322203.1, NP_001322205.1, NP_001322204.1, NP_001117266.1
Others	UniRef100: UniRef100_O04093 UniRef90: UniRef90_O04093 UniRef50: UniRef50_O04093 UniGene: At.28785

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mRNA

Protein

UniprotKB

Description

Refseq

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全选

Pubmed编号	文献信息	发表日期	相关基因
23634836	Characterization of the LOV1-mediated, victorin-induced, cell-death response with virus-induced gene silencing. Mol. Plant Microbe Interact. Brian M Gilbert , Thomas J Wolpert	2013-08-01	LOV1
23087001	Tricking the guard: exploiting plant defense for disease susceptibility. Science J Lorang , T Kidarsa , C S Bradford , B Gilbert , M Curtis , S-C Tzeng , C S Maier , T J Wolpert	2012-11-02	-
19880798	Domain swapping to assess the mechanistic basis of Arabidopsis phototropin 1 receptor kinase activation and endocytosis by blue light. Plant Cell Eirini Kaiserli , Stuart Sullivan , Matthew A Jones , Kevin A Feeney , John M Christie	2009-10-01	-
18607090	Crystallization and preliminary X-ray diffraction analysis [correction of anaylsis] of the LOV1 domains of phototropin 1 and 2 from Arabidopsis thaliana. Acta Crystallogr. Sect. F Struct. Biol. Cryst. Commun. Masayoshi Nakasako , Michihiro Hirata , Nobutaka Shimizu , Shyuntaro Hosokawa , Daisuke Matsuoka , Toshihiko Oka , Masaki Yamamoto , Satoru Tokutomi	2008-07-01	-
18052878	Characterization of natural and induced variation in the LOV1 gene, a CC-NB-LRR gene conferring victorin sensitivity and disease susceptibility in Arabidopsis. Mol. Plant Microbe Interact. Teresa A Sweat , Jennifer M Lorang , Erica G Bakker , Thomas J Wolpert	2008-01-01	LOV1
17804803	Plant disease susceptibility conferred by a "resistance" gene. Proc. Natl. Acad. Sci. U.S.A. Jennifer M Lorang , Teresa A Sweat , Thomas J Wolpert	2007-09-11	LOV1
17653269	Control of flowering time and cold response by a NAC-domain protein in Arabidopsis. PLoS ONE So Yeon Yoo , Yunhee Kim , Soo Young Kim , Jong Seob Lee , Ji Hoon Ahn	2007-01-01	NAC2, LOV1, NAC1
11130712	Sequence and analysis of chromosome 1 of the plant Arabidopsis thaliana. Nature A Theologis , J R Ecker , C J Palm , N A Federspiel , S Kaul , O White , J Alonso , H Altafi , R Araujo , C L Bowman , S Y Brooks , E Buehler , A Chan , Q Chao , H Chen , R F Cheuk , C W Chin , M K Chung , L Conn , A B Conway , A R Conway , T H Creasy , K Dewar , P Dunn , P Etgu , T V Feldblyum , J Feng , B Fong , C Y Fujii , J E Gill , A D Goldsmith , B Haas , N F Hansen , B Hughes , L Huizar , J L Hunter , J Jenkins , C Johnson-Hopson , S Khan , E Khaykin , C J Kim , H L Koo , I Kremenetskaia , D B Kurtz , A Kwan , B Lam , S Langin-Hooper , A Lee , J M Lee , C A Lenz , J H Li , Y Li , X Lin , S X Liu , Z A Liu , J S Luros , R Maiti , A Marziali , J Militscher , M Miranda , M Nguyen , W C Nierman , B I Osborne , G Pai , J Peterson , P K Pham , M Rizzo , T Rooney , D Rowley , H Sakano , S L Salzberg , J R Schwartz , P Shinn , A M Southwick , H Sun , L J Tallon , G Tambunga , M J Toriumi , C D Town , T Utterback , S Van Aken , M Vaysberg , V S Vysotskaia , M Walker , D Wu , G Yu , C M Fraser , J C Venter , R W Davis	2000-12-14	AT1G27045, AT1G24095, AT1G12855, AT1G63295, AT1G45163, AT1G20735, AT1G51035, AT1G55207, AT1G25145, AT1G53023, AT1G29025, AT1G19396, AT1G26515, AT1G78476, AT1G71866, AT1G36922, AT1G47578, AT1G11125, AT1G02575, AT1G25150, AT1G62914, AT1G51020, AT1G03365, AT1G53025, AT1G50732, AT1G09794, AT1G64583, AT1G05675, AT1G33612, AT1G03103, AT1G03106, AT1G05085, AT1G06135, AT1G06475, DSI-1VOC, AT1G07730, AT1G07985, AT1G08125, AT1G12805, AT1G13635, AT1G16515, AT1G17285, AT1G18265, AT1G19394, AT1G19397, AT1G21395, AT1G23465, AT1G23935, AT1G24580, AT1G26798, AT1G26799, AT1G28695, AT1G30475, AT1G31835, AT1G43666, AT1G45165, AT1G48195, AT1G48325, ATHAL3B, AT1G52315, CDT1, AT1G52855, AT1G52905, AT1G53285, AT1G53785, AT1G53815, AT1G54217, AT1G54575, AT1G55475, AT1G55675, AT1G56045, AT1G56415, AT1G58242, AT1G59865, SCRL5, AT1G61097, AT1G61105, BRD4, AT1G62085, AT1G63055, AT1G64065, AT1G64235, AT1G64255, AT1G72645, AT1G76955, AT1G77885, GL22, AT1G03495, AT1G03982, AT1G04625, AT1G05894, AT1G06645, AT1G06923, YAB2, SCRL3, AT1G09157, AT1G10417, AT1G10850, AT1G12064

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