| Type | Description |
|---|---|
| Definition | breaking of asymmetry in the stomatal lineage |
| Date | Results | Publications |
|---|---|---|
| 2019-10-19 11:04:00 | Ectopic induction of the stomatal protein BASL (BREAKING OF ASYMMETRY IN THE STOMATAL LINEAGE) reveals a tissue-wide epidermal polarity field in leaves throughout development. | 30100337 |
| 2017-12-30 10:51:00 | BASL polarization leads to elevated nuclear MPK6 signaling and lowered SPCH abundance in one of the two daughter cells. Therefore, two daughter cells are differentiated by BASL polarity-mediated differential suppression of SPCH, which may provide developmental plasticity in plant stem cell asymmetric cell division. | 27746029 |
| 2017-11-11 10:40:00 | fine-scale dissection of the subdomains of polarity in stomatal asymmetric cell division | 27422992 |
| 2015-08-01 11:24:00 | a positive-feedback loop between BASL and the MAPK pathway constitutes a polarity module at the cortex. Cortical localization of BASL requires phosphorylation mediated by MPK3/6. | 25843888 |
| 2010-01-21 00:00:00 | Study identified BASL, a novel regulator of asymmetric divisions in Arabidopsis. | 19523675 |
| Type | IDs |
|---|---|
| Synonymous | BREAKING OF ASYMMETRY IN THE STOMATAL LINEAGE, MAE1.13, MAE1_13 |
| Gene |
UniProtKB-ID:
BASL_ARATH,
A0A5S9YFY4_ARATH
UniprotKB:
Q5BPF3,
A0A5S9YFY4
UniParc:
UPI00001628ED,
UPI00005DC185
EMBL:
AY800654,
AB008269,
AB015472,
AY924875,
CP002688,
AY800653,
CACSHJ010000096
EnsemblGenome:
AT5G60880
KO:
ath:AT5G60880
|
| Nucleutide sequences |
EMBL-CDS:
BAB10108.1,
AAX23950.1,
AED97391.1,
AAV68889.1,
AED97392.1,
AAV68890.1,
CAA0411173.1
Gene_ORFName:
MAE1.13,
C24_LOCUS26216
EnsemblGenome_TRS:
AT5G60880.1,
AT5G60880.2
|
| Protein sequencees |
EnsemblGenome_PRO:
AT5G60880.2,
AT5G60880.1
RefSeq:
NP_001032115.1,
NP_200896.1
|
| Others |
UniRef100:
UniRef100_Q5BPF3
UniRef90:
UniRef90_Q5BPF3
UniRef50:
UniRef50_Q5BPF3
UniGene:
At.51260
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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