Type | Description |
---|---|
Definition | Disease resistance protein (TIR-NBS-LRR class) |
Date | Results | Publications |
---|---|---|
2016-07-16 11:01:00 | The disruption of At5g11250/BURNOUT1 (BNT1) causes a drastic increase of the jasmonic, salicylic, and abscisic acid, as well as ethylene, levels. The knock-out bnt1 mutants displayed a multitude of growth and development defects. | 26398806 |
Type | IDs |
---|---|
Synonymous | F2I11.140, F2I11_140 |
Gene |
UniProtKB-ID:
Q9LFN1_ARATH,
A0A1P8BFM2_ARATH,
A0A654G0B9_ARATH
UniprotKB:
Q9LFN1,
A0A1P8BFM2,
A0A654G0B9
UniParc:
UPI00000A4F8E,
UPI000848A773
EMBL:
CP002688,
CACRSJ010000110,
AL360314
EnsemblGenome:
AT5G11250
KO:
ath:AT5G11250
|
Nucleutide sequences |
EMBL-CDS:
CAB96660.1,
ANM70414.1,
AED91652.2,
ANM70413.1,
VYS66517.1
Gene_ORFName:
F2I11.140,
F2I11_140,
F2I11.140,
F2I11_140,
AN1_LOCUS21919
|
Protein sequencees |
RefSeq:
NP_001332026.1,
NP_196686.1,
NP_001318538.1
|
Others |
UniRef100:
UniRef100_Q9LFN1
UniRef90:
UniRef90_Q9LFN1
UniRef50:
UniRef50_Q9LFN1
UniGene:
At.54815
|
{{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
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Refseq |
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