例如:"NBL1", "4681", "drought"
收藏
81532 MOB2

81532

MOB2

MOB kinase activator 2

protein-coding

Homo sapiens

基因描述

Type Description
Definition MOB kinase activator 2

研究结论

Date Results Publications
2021-04-13 10:11:00 MOB2 suppresses GBM cell migration and invasion via regulation of FAK/Akt and signaling. 32286266
2015-12-19 12:02:00 The altered cell-matrix adhesion and cell motility induced by hMOB2 expression was caused by the assembly of focal adhesions. 25779224
2015-08-29 10:28:00 hMOB2 interacts with RAD50, facilitating the recruitment of the MRE11-RAD50-NBS1 (MRN) DNA damage sensor complex and activated ATM to DNA damaged chromatin. 25460043
2014-01-11 10:33:00 MOB2 overexpression induces apoptosis and inhibits the growth of human hepatic cancer cells 23326134
2010-12-05 22:08:00 Observational study of gene-disease association, gene-gene interaction, and gene-environment interaction. (HuGE Navigator) 20889853

名称对应

Type IDs
Synonymous HCCA2
Gene
UniProtKB-ID: MOB2_HUMAN
UniprotKB: Q70IA6
UniParc: UPI00001C1575, UPI000049811C, UPI00000373CD
EMBL: BC067785, AC091196, AK296658, BC047291, AK057350, AJ580639
Ensembl: ENSG00000182208
KO: hsa:81532
Nucleutide sequences
EMBL-CDS: CAE45271.1, BAG59255.1, AAH47291.1, BAB71443.1, AAH67785.1
Ensembl_TRS: ENST00000329957
Protein sequencees
Ensembl_PRO: ENSP00000328694
RefSeq: NP_443731.2, NP_001165694.1
Others
UniRef100: UniRef100_Q70IA6
UniRef90: UniRef90_Q70IA6
UniRef50: UniRef50_Q70IA6
UniGene: Hs.370360
CCDS: CCDS53591.1

全选

{{proteinIndex+1}}
mRNA Protein UniprotKB Description
Refseq {{protein.nucleotideVersion}}

Ensembl: {{protein.nucleotideEnsembl}}

{{protein.proteinVersion}}

Ensembl: {{protein.proteinEnsembl}}

{{uniprot}}
Definition: {{{protein.definition}}}

Transcript Veriant:{{protein.transcriptVeriant}}

Status: {{protein.status}}

Location ( {{protein.positionStart}}..{{protein.positionEnd}} )

暂无数据

研究热度

热点单位
排名 科研单位 文献
{{affIndex+1}}

{{aff.value}}

{{aff.size}}

目前还没有研究热点单位

热点人员
排名 研究人员 文献
{{authorIndex+1}}

{{author.value}}

{{author.size}}

目前还没有研究热点人员