| Type | Description |
|---|---|
| Definition | ATPase type 13A2 |
| Date | Results | Publications |
|---|---|---|
| 2019-03-23 10:35:00 | Study shows for the first time in vivo that loss of function of ATP13A2 causes an increased sensitivity to manganese. | 28595912 |
| 2019-03-09 10:23:00 | Loss of one functional Atp13a2 allele leads to selective gliosis independent of robust lipofuscinosis. | 29859891 |
| 2013-11-02 10:17:00 | the loss of Atp13a2 causes sensorimotor impairments, alpha-synuclein accumulation as occurs in PD and related synucleinopathies, and accumulation of lipofuscin deposits characteristic of NCL | 23393156 |
| 2012-06-23 11:59:00 | study reveals a number of intriguing neuronal phenotypes due to the loss- or gain-of-function of ATP13A2 that support a role for this protein in regulating intracellular cation homeostasis and neuronal integrity | 22186024 |
| 2012-06-09 11:47:00 | This study showed that ATP13A2 regulates mitochondrial bioenergetics through macroautophagy. | 22198378 |
| Type | IDs |
|---|---|
| Synonymous | 1110012E06Rik, AA589443 |
| Gene |
UniProtKB-ID:
AT132_MOUSE,
E9Q2A4_MOUSE
UniprotKB:
Q9CTG6,
E9Q2A4
UniParc:
UPI0001B2B403,
UPI00001C31A1
EMBL:
AK003623,
AL645625,
BC042661
Ensembl:
ENSMUSG00000036622
KO:
mmu:74772
|
| Nucleutide sequences |
EMBL-CDS:
BAB22896.1,
AAH42661.1
Ensembl_TRS:
ENSMUST00000037055,
ENSMUST00000127833
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000039648,
ENSMUSP00000132183
RefSeq:
NP_001366551.1,
XP_006539299.1,
NP_001366548.1,
NP_001366549.1,
NP_001157838.1,
NP_001366550.1,
XP_036020402.1,
XP_006539300.1,
NP_083373.2
|
| Others |
UniRef100:
UniRef100_Q9CTG6,
UniRef100_E9Q2A4
UniRef90:
UniRef90_Q9CTG6
UniRef50:
UniRef50_Q9CTG6
UniGene:
Mm.205625
CCDS:
CCDS18859.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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