| Type | Description |
|---|---|
| Definition | hydrogen voltage-gated channel 1 |
| Date | Results | Publications |
|---|---|---|
| 2021-01-09 13:31:00 | Loss of voltage-gated proton channel Hv1 leads to diet-induced obesity in mice. | 32049639 |
| 2020-11-21 13:21:00 | Hv1/VSOP regulates neutrophil directional migration and ERK activity by tuning production. | 32303121 |
| 2020-05-02 10:56:00 | Hv1-deficiency protects beta cells from high glucose(HG)treatment-induced damage. Hv1-deficiency protects beta cells by reducing HG-induced reactive oxygen species production. The high level in HG-treatment beta cells may owe to NOX4 upregulation. HV1-deficiency inhibits the HG-treatment induced NOX4 upregulation in beta cells. | 30967259 |
| 2019-08-24 12:55:00 | The increase in production in VSOP/Hv1-deficient cells was not observed in macrophages, which suggests microglia have a unique mechanism of VSOP/Hv1-dependent duanyu1670 regulation. | 28961043 |
| 2019-07-06 10:44:00 | This work therefore demonstrates that fully differentiated mammalian muscle fibers do not express functional voltage-gated H(+) channels and consequently can no longer be considered as the only vertebrate excitable cells exhibiting voltage-gated H(+) currents. | 30281323 |
| Type | IDs |
|---|---|
| Synonymous | 0610039P13Rik, AI450555, BTS, HV1, Vsop., mVSOP |
| Gene |
UniProtKB-ID:
HVCN1_MOUSE
UniprotKB:
Q3U2S8
UniParc:
UPI000002858E
EMBL:
AK002854,
AK170422,
BC021548,
AK155123,
AK154880
Ensembl:
ENSMUSG00000064267
KO:
mmu:74096
|
| Nucleutide sequences |
EMBL-CDS:
BAE41787.1,
AAH21548.1,
BAE32899.1,
BAB22409.1,
BAE33062.1
Ensembl_TRS:
ENSMUST00000072602,
ENSMUST00000100747
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000098312,
ENSMUSP00000072401
RefSeq:
NP_083028.1,
XP_006530535.1,
XP_006530534.1,
NP_001346383.1,
NP_001035954.1
|
| Others |
UniRef100:
UniRef100_Q3U2S8
UniRef90:
UniRef90_Q3U2S8
UniRef50:
UniRef50_Q96D96-3
UniGene:
Mm.28804
CCDS:
CCDS19644.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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