Type | Description |
---|---|
Definition | TRAF3 interacting protein 1 |
Date | Results | Publications |
---|---|---|
2012-01-07 12:45:00 | Homozygous Traf3ip1 mutants are not viable and have both neural developmental defects and polydactyly, phenotypes typical of mutants with ciliary assembly defects. | 21945076 |
2010-01-21 00:00:00 | Data show that overexpression of IFT54/Traf3ip1 displaces IFT20 from the Golgi apparatus. IFT54s effect on IFT20 is a dominant negative phenotype caused by its overexpression. | 19253336 |
Type | IDs |
---|---|
Synonymous | 3930402D05Rik, AU041749, MIP-T3 |
Gene |
UniProtKB-ID:
MIPT3_MOUSE,
A0A087WQD8_MOUSE
UniprotKB:
Q149C2,
A0A087WQD8
UniParc:
UPI0003D73E4C,
UPI00001C22C9
EMBL:
AY613437,
AK014457,
BC117868,
AC109199,
BC046538
Ensembl:
ENSMUSG00000034292
KO:
mmu:74019
|
Nucleutide sequences |
EMBL-CDS:
AAT72918.1,
BAB29364.1,
AAH46538.1,
AAI17869.1
Ensembl_TRS:
ENSMUST00000047242,
ENSMUST00000189341
|
Protein sequencees |
Ensembl_PRO:
ENSMUSP00000042391,
ENSMUSP00000140151
RefSeq:
XP_006529961.1,
NP_082994.1,
XP_036009760.1,
XP_006529959.1,
XP_036009757.1
|
Others |
UniRef100:
UniRef100_Q149C2,
UniRef100_A0A087WQD8
UniRef90:
UniRef90_Q149C2
UniRef50:
UniRef50_Q8TDR0
UniGene:
Mm.318430,
Mm.491399
CCDS:
CCDS35664.1
|
排名 | 科研单位 | 文献 |
---|---|---|
1 |
University of Massachusetts Medical School |
2 |
2 |
Yokohama Institute |
1 |
3 |
RIKEN Yokohama Institute 1-7-22 Suehiro-cho |
1 |
4 |
RIKEN Yokohama Institute |
1 |
5 |
Department of Genetics, Harvard Medical School, Boston, Massachusetts, USA. |
1 |
排名 | 研究人员 | 文献 |
---|---|---|
1 |
P Carninci |
7 |
2 |
Y Hayashizaki |
6 |
3 |
K Shibata |
5 |
4 |
M Itoh |
5 |
5 |
H Konno |
5 |