Type | Description |
---|---|
Definition | Ras-related associated with diabetes |
Date | Results | Publications |
---|---|---|
2020-07-11 12:46:00 | Compared to normal adipocytes, slincRAD knockdown cells had defective differentiation features, such as smaller sizes and decreased lipid production. For elder mice, slincRAD knockdown led to abnormal glucose and lipid metabolism. | 31005260 |
2018-05-19 10:36:00 | we report that Rad GTPase function is required for normal bone homeostasis in mice | 28732776 |
2017-10-28 12:11:00 | Rad reduction provides a stable inotropic response rooted in sarcomere level function in hearts of knockout mice. | 27798760 |
2016-02-06 11:38:00 | The Rad(-/-) phenotype is marked by enhanced systolic and diastolic function, which is consistent with a model that does not progress into heart failure. | 26371164 |
2015-06-20 10:51:00 | Rad, Gem and Rem all reduce depolarization-dependent Ca2+ entry in developing myotubes. | 24476902 |
Type | IDs |
---|---|
Synonymous | REM3, Rad |
Gene |
UniProtKB-ID:
Q6PGA2_MOUSE
UniprotKB:
Q6PGA2
UniParc:
UPI00000238F1
EMBL:
AC138617,
AK159550,
BC057138
Ensembl:
ENSMUSG00000031880
KO:
mmu:56437
|
Nucleutide sequences |
EMBL-CDS:
BAE35175.1,
AAH57138.1
Ensembl_TRS:
ENSMUST00000034351
|
Protein sequencees |
Ensembl_PRO:
ENSMUSP00000034351
RefSeq:
NP_062636.2,
XP_006531269.1
|
Others |
UniRef100:
UniRef100_Q6PGA2
UniRef90:
UniRef90_O88667
UniRef50:
UniRef50_O88667
UniGene:
Mm.29467
|
{{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
---|---|---|---|---|---|---|---|---|
Refseq |
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