| Type | Description |
|---|---|
| Definition | odontogenic, ameloblast associated |
| Date | Results | Publications |
|---|---|---|
| 2020-02-08 10:52:00 | AMELX and ODAM variations was not different between two populations of schoolchildren with respect to dental fluorosis (DF) severity; however, the presence of rs1784418 differed between phenotypes with regard to susceptibility to DF. Therefore, MMP20 might be related to the various phenotypes of DF | 31838295 |
| 2019-06-22 11:20:00 | Similar to AMTN, ODAM is able to promote HA nucleation in a dose-dependent manner in SBF, and even outside of its biological context in vitro. | 29745811 |
| 2017-06-24 11:15:00 | Risk of recurrence correlated with cytoplasmic expression of ODAM and pAKT, whereas nuclear expression did not predict recurrence of gastrointestinal stromal tumors. | 27229116 |
| 2017-02-18 10:06:00 | The inhibitory effects of ODAM on colorectal cancer cell growth were associated with PTEN elevation and PI3K/AKT signaling inactivation. | 27694004 |
| 2016-08-27 11:51:00 | ODAM induced RhoA activity. ODAM-mediated RhoA signalling resulted in actin filament rearrangement, increased cell adhesion and inhibited the migration/invasion of MCF7 cells. These results suggest that ODAM expression maintains their adhesion, resulting in the prevention of their metastasis via the regulation of RhoA signalling in breast cancer cells. | 26358398 |
| Type | IDs |
|---|---|
| Synonymous | APIN |
| Gene |
UniProtKB-ID:
ODAM_HUMAN
UniprotKB:
A1E959
UniParc:
UPI0000413A02
EMBL:
CH471057,
AK000520,
BC017796,
EF113908
Ensembl:
ENSG00000109205
KO:
hsa:54959
|
| Nucleutide sequences |
EMBL-CDS:
ABL11577.1,
EAX05612.1,
BAA91226.1,
AAH17796.1
Ensembl_TRS:
ENST00000396094
|
| Protein sequencees |
Ensembl_PRO:
ENSP00000379401
RefSeq:
XP_016863820.1,
NP_060325.3,
NP_001372508.1
|
| Others |
UniRef100:
UniRef100_A1E959
UniRef90:
UniRef90_A1E959
UniRef50:
UniRef50_A1E959
UniGene:
Hs.143811
CCDS:
CCDS3536.2
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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