Type | Description |
---|---|
Definition | contactin 6 |
Date | Results | Publications |
---|---|---|
2020-10-31 13:18:00 | Time origin and structural analysis of the induced CRISPR/cas9 megabase-sized deletions and duplications involving the Cntn6 gene in mice. | 31578377 |
2020-04-11 12:03:00 | NB-3 deficiency promoted synapse reformation between sRST regenerative axons and motor neurons and enhanced the potential for electrical activity of muscle contraction and motor coordination. | 30156464 |
2019-12-21 12:20:00 | decrease of vermian SYP1 and CNTN6 in reln(+/-) mice displayed patterns compatible with the structural modifications of the autistic cerebellum. | 31098770 |
2019-03-09 12:02:00 | Cntn6 is expressed during postnatal hippocampal development. The absence of Cntn6 affects hippocampal Spatial Learning and memory, and affects allocentric navigation of the animals. | 30106251 |
2018-02-10 11:15:00 | The common phenotypic features of 40 patients with microdeletions and microduplications of CNTN6 appeared to be the autism spectrum disorders, developmental delay, intellectual disability, seizures, cognitive impairment, cardiological defects, and behavioral problems. | 29369566 |
Type | IDs |
---|---|
Synonymous | NB-3 |
Gene |
UniProtKB-ID:
CNTN6_MOUSE
UniprotKB:
Q9JMB8
UniParc:
UPI00000E9AAE,
UPI00004650A7
EMBL:
AK052972,
AB032602,
BC076594
Ensembl:
ENSMUSG00000030092
KO:
mmu:53870
|
Nucleutide sequences |
EMBL-CDS:
BAA92367.1,
BAC35227.1,
AAH76594.1
Ensembl_TRS:
ENSMUST00000089215,
ENSMUST00000162872
|
Protein sequencees |
Ensembl_PRO:
ENSMUSP00000124025,
ENSMUSP00000086623
RefSeq:
XP_036022181.1,
XP_036022178.1,
XP_017177167.1,
XP_036022177.1,
XP_036022179.1,
XP_006506446.1,
XP_006506447.1,
XP_036022184.1,
XP_006506445.1,
NP_059079.2,
XP_036022183.1,
XP_036022185.1,
XP_036022180.1,
XP_036022182.1,
XP_011239711.1,
XP_006506444.1,
XP_017177168.1
|
Others |
UniRef100:
UniRef100_Q9JMB8
UniRef90:
UniRef90_Q9JMB8
UniRef50:
UniRef50_Q9JMB8
UniGene:
Mm.321671
CCDS:
CCDS20394.1
|
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