| Type | Description |
|---|---|
| Definition | G0/G1 switch 2 |
| Date | Results | Publications |
|---|---|---|
| 2021-04-17 13:15:00 | Low G0S2 gene expression levels in peripheral blood may be a genetic marker of acute myocardial infarction in patients with stable coronary atherosclerotic disease: A retrospective clinical study. | 33545927 |
| 2020-07-11 11:43:00 | G0S2 hypermethylation is a hallmark of rapidly recurrent or fatal ACC, amenable to targeted assessment using routine molecular diagnostics. Assessing G0S2 methylation is straightforward, feasible for clinical decision-making, and will enable the direction of efficacious adjuvant therapies for patients with aggressive ACC. | 30770352 |
| 2020-05-16 11:37:00 | In addition to its role as a lipolytic inhibitor, G0S2 is capable of directly promoting TG synthesis by acting as a lipid-synthesizing enzyme. | 30802154 |
| 2020-05-09 11:08:00 | We conclude that the RNF126/BAG6 complex contributes to G0S2 degradation and that interventions to prevent G0S2 degradation may offer a therapeutic strategy for managing ischemic diseases. | 31371451 |
| 2018-08-11 12:01:00 | ER+ breast cancer cells with restored G0S2 expression had a relative increased sensitivity to tamoxifen | 28910567 |
| Type | IDs |
|---|---|
| Gene |
UniProtKB-ID:
G0S2_HUMAN
UniprotKB:
P27469
UniParc:
UPI000012AE19
EMBL:
AL031316,
CH471100,
M72885,
BT007101,
CR542179,
M69199,
CR542193,
BC009694
Ensembl:
ENSG00000123689
KO:
hsa:50486
|
| Nucleutide sequences |
EMBL-CDS:
CAG46976.1,
AAB04044.1,
CAG46990.1,
AAP35765.1,
AAH09694.1,
AAA58966.1,
EAW93447.1
Ensembl_TRS:
ENST00000367029
|
| Protein sequencees |
Ensembl_PRO:
ENSP00000355996
RefSeq:
NP_056529.1
|
| Others |
UniRef100:
UniRef100_P27469
UniRef90:
UniRef90_P27469
UniRef50:
UniRef50_P27469
UniGene:
Hs.432132
CCDS:
CCDS1488.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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