| Type | Description |
|---|---|
| Definition | tafazzin |
| Date | Results | Publications |
|---|---|---|
| 2019-10-26 10:28:00 | Results find that knock-out of tafazzin (Taz) changed substantially the profile of fatty acids incorporated in cardiolipin (CL) and the distribution of CL species. It was further associated with decreased capacity of oxidative phosphorylation and switch to glycolysis, altered morphology and arrangement, and reduction in glioma cell proliferation. | 30639735 |
| 2018-06-02 11:55:00 | Knock-down of tafazzin had no consequence with respect to the pattern of cellular fatty acids but caused a decrease in cell proliferation. | 29325722 |
| 2013-12-28 10:44:00 | Tafazzin knockdown interrupts the neonatal ventricular fibroblast cell cycle and this in turn may contribute to fibrosis and dilated cardiomyopathy in Barth syndrome. | 23997105 |
| 2010-07-12 11:39:00 | Data support the concept that a decreased tafazzin expression causes cardiomyocyte hypertrophy in vitro. | 20348225 |
| Type | IDs |
|---|---|
| Gene |
UniProtKB-ID:
Q4KLG6_RAT
UniprotKB:
Q4KLG6
UniParc:
UPI0000180AC3
EMBL:
CH474099,
BC099221,
AC094668
Ensembl:
ENSRNOG00000053023
KO:
rno:363521
|
| Nucleutide sequences |
EMBL-CDS:
AAH99221.1,
EDL84983.1
Gene_ORFName:
rCG_43825
Ensembl_TRS:
ENSRNOT00000089778
|
| Protein sequencees |
Ensembl_PRO:
ENSRNOP00000071612
RefSeq:
XP_017457607.1,
XP_006229661.1,
XP_006229662.1,
NP_001020919.1
|
| Others |
UniRef100:
UniRef100_Q4KLG6
UniRef90:
UniRef90_Q4KLG6
UniRef50:
UniRef50_Q4KLG6
UniGene:
Rn.7267
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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