| Type | Description |
|---|---|
| Definition | tafazzin |
| Date | Results | Publications |
|---|---|---|
| 2018-12-01 11:34:00 | Disrupted gene function in yap1(-/-); taz(+/-) embryos did not disturb liver bud formation, but instead impaired cell proliferation in liver and movement of the neighboring lateral plate mesoderm (LPM). Overexpression of wild type yap1 or taz could rescue the defective liver phenotypes in yap1(-/-); taz(+/-) embryos. | 29859190 |
| 2017-09-09 11:31:00 | During vascular regression, Yap/Taz is activated by blood circulation in the endothelial cells. This leads to induction of Ctgf and actin polymerization. Interference with Yap/Taz activation decreased Ctgf production, which decreased actin polymerization and vascular regression. | 28369143 |
| 2010-01-21 00:00:00 | knockdown phenotype demonstrates that abnormal cardiac development, with a linear, nonlooped heart, and hypomorphic tail and eye development proves that tafazzin is essential for overall zebrafish development, especially of the heart. | 16794186 |
| Type | IDs |
|---|---|
| Synonymous | wu:fb39f12, zgc:91803 |
| Gene |
UniProtKB-ID:
Q6PUS0_DANRE
UniprotKB:
Q6PUS0
UniParc:
UPI000035A86B
EMBL:
AY576995
KO:
dre:321965
|
| Nucleutide sequences |
EMBL-CDS:
AAS92633.1
|
| Protein sequencees |
RefSeq:
XP_021325408.1,
XP_009294889.1,
NP_001001814.1
|
| Others |
UniRef100:
UniRef100_Q6PUS0
UniRef90:
UniRef90_F1QCP6
UniRef50:
UniRef50_Q4KLG6
UniGene:
Dr.3504
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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