| Type | Description |
|---|---|
| Definition | YME1-like 1 (S. cerevisiae) |
| Date | Results | Publications |
|---|---|---|
| 2019-08-10 12:25:00 | YME1L-deficient mice manifest ocular dysfunction with microphthalmia and cataracts and develop deficiencies in locomotor activity due to specific degeneration of spinal cord axons, which relay proprioceptive signals from the hind limbs to the cerebellum. | 30389680 |
| 2016-07-02 11:20:00 | Data suggest that in a mouse model of neonatal hypoxic-ischemic brain injury, the expression of mitochondrial shaping proteins, such as OPA1 and Yme1L, are altered; in vitro and in vivo, OPA1 is cleaved to shorter forms and Yme1L expression is reduced. | 26393574 |
| 2014-05-31 10:04:00 | Constitutive OPA1 cleavage by YME1L and OMA1 at two distinct sites leads to the accumulation of both long and short forms of OPA1 and maintains mitochondrial fusion. | 24616225 |
| Type | IDs |
|---|---|
| Synonymous | FtsH1, Ftsh |
| Gene |
UniProtKB-ID:
YMEL1_MOUSE
UniprotKB:
O88967
UniParc:
UPI00000229E4
EMBL:
AY136286,
AF090430,
AY136287,
BC007128,
AK079175
Ensembl:
ENSMUSG00000026775
KO:
mmu:27377
|
| Nucleutide sequences |
EMBL-CDS:
BAC37568.1,
AAN17725.1,
AAN17724.1,
AAC35558.1,
AAH07128.1
Ensembl_TRS:
ENSMUST00000028117
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000028117
RefSeq:
NP_038799.1
|
| Others |
UniRef100:
UniRef100_O88967
UniRef90:
UniRef90_O88967
UniRef50:
UniRef50_Q96TA2
UniGene:
Mm.23335
CCDS:
CCDS15728.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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