Type | Description |
---|---|
Definition | methyltransferase like 14 |
Date | Results | Publications |
---|---|---|
2021-03-20 13:33:00 | N(6)-Adenosine Methylation of Socs1 mRNA Is Required to Sustain the Negative Feedback Control of Macrophage Activation. | 33220174 |
2021-03-20 13:25:00 | The N6-methyladenosine mRNA methylase METTL14 promotes renal ischemic reperfusion injury via suppressing YAP1. | 31318098 |
2020-05-09 12:57:00 | METTL14 deficiency in beta-cells increased cell death, altered cell differentiation, and decreased beta-cell mass and insulin secretion, leading to glucose intolerance. Mettl14 deletion is due to unique functions of m6A methylation in beta-cells mediated through other reader proteins or caused by secondary effects following chronically altered beta-cell functions. | 31029827 |
2020-04-04 11:42:00 | conditional inactivation of an essential N(6)-methyladenosine writer component, METTL14, results in decreased oligodendrocyte numbers and CNS hypomyelination, although oligodendrocyte precursor cell numbers are normal. | 31901304 |
2019-12-07 10:38:00 | These results suggest that acute METTL14 deficiency in beta-cells induces glucose intolerance by increasing the IRE1alpha/sXBP-1 pathway. | 31369074 |
Type | IDs |
---|---|
Synonymous | G430022H21Rik, mKIAA1627 |
Gene |
UniProtKB-ID:
MET14_MOUSE
UniprotKB:
Q3UIK4
UniParc:
UPI00001C2D8A,
UPI000166301A
EMBL:
AK146881,
BC052204,
AK129409
Ensembl:
ENSMUSG00000028114
KO:
mmu:210529
|
Nucleutide sequences |
EMBL-CDS:
AAH52204.1,
BAC98219.1,
BAE27502.1
Ensembl_TRS:
ENSMUST00000029759
|
Protein sequencees |
Ensembl_PRO:
ENSMUSP00000029759
RefSeq:
NP_964000.2
|
Others |
UniRef100:
UniRef100_Q3UIK4
UniRef90:
UniRef90_Q9HCE5
UniRef50:
UniRef50_Q9HCE5
UniGene:
Mm.260039
CCDS:
CCDS38622.1
|
{{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
---|---|---|---|---|---|---|---|---|
Refseq |
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