| Type | Description |
|---|---|
| Definition | msh homeobox 2 |
| Date | Results | Publications |
|---|---|---|
| 2020-07-18 12:14:00 | Loss of Msx1 and Msx2 in neural stem cells lead the small size of the brain in mice. | 31221479 |
| 2020-07-11 12:08:00 | Used Msx2 conditional knockouts as a model and found that Msx2 deficiency in surface ectoderm induced ocular anterior segment dysgenesis. Loss of Msx2 function specifically affected lens development, while other eye structures were not significantly affected. Multiple lines of evidence show that calcium signaling pathways are involved in this pathogenesis. | 30683901 |
| 2020-06-13 13:05:00 | miR-203 overexpression promoted proliferation, migration, and osteogenic differentiation of MC3T3-E1 cells through upregulating Msx2 along with activation of the pathways | 30854680 |
| 2019-12-07 10:22:00 | When conditional, tissue-specific ablation of both Msx1 and Msx2 was accomplished in the mouse uterus, a dramatically impaired decidual response was observed. In the absence of both Msx1 and Msx2, uterine stromal cells were able to proliferate, but they failed to undergo terminal differentiation | 31125045 |
| 2019-09-07 10:23:00 | marks spatially restricted populations of mesenchymal precursor cells with diverse differentiation potential | 29746183 |
| Type | IDs |
|---|---|
| Synonymous | BB122635, Hox-8, Hox8, Hox8.1 |
| Gene |
UniProtKB-ID:
MSX2_MOUSE,
Q3UZH5_MOUSE
UniprotKB:
Q03358,
Q3UZH5
UniParc:
UPI0000021C03
EMBL:
S60698,
AK131974,
X59252,
BC141132,
L11739,
M38576,
L11738,
S60460,
AK133846
Ensembl:
ENSMUSG00000021469
KO:
mmu:17702
|
| Nucleutide sequences |
EMBL-CDS:
AAA37824.1,
AAA03477.1,
CAA41945.1,
AAC60657.2,
BAE21882.1,
AAI41133.1,
BAE20914.1
Ensembl_TRS:
ENSMUST00000021922
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000021922
RefSeq:
NP_038629.2
|
| Others |
UniRef100:
UniRef100_Q03358
UniRef90:
UniRef90_Q03358
UniRef50:
UniRef50_Q03358
UniGene:
Mm.1763
CCDS:
CCDS26522.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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