| Type | Description |
|---|---|
| Definition | MaX-Like |
| Date | Results | Publications |
|---|---|---|
| 2017-11-26 01:38:00 | The Mad-like transcription factor MDL-1, and the Max-like transcription factors MXL-1 and MXL-2 promote pathogen levels, while the Myc-Mondo-like transcription factor MML-1 inhibits pathogen levels. We used epistasis analysis to show that MDL-1 and MXL-1, which are thought to function as a heterodimer, appear to be acting canonically. | 27402359 |
| 2010-01-21 00:00:00 | MondoA and Myc may have more predominant roles in cell migration than previously appreciated, and their cooperation with other pro-migratory pathways provides a more integrated view of their role in cell migration. | 17826759 |
| Type | IDs |
|---|---|
| Gene |
UniProtKB-ID:
Q9TZ70_CAEEL
UniprotKB:
Q9TZ70
UniParc:
UPI0000076903
EMBL:
BX284603
EnsemblGenome:
WBGene00003510
KO:
cel:CELE_F40G9.11
|
| Nucleutide sequences |
EMBL-CDS:
CCD70935.1
Gene_ORFName:
CELE_F40G9.11,
F40G9.11
|
| Protein sequencees |
RefSeq:
NP_497173.1
|
| Others |
UniRef100:
UniRef100_Q9TZ70
UniRef90:
UniRef90_Q9TZ70
UniRef50:
UniRef50_Q9TZ70
UniGene:
Cel.10506
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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