| Type | Description |
|---|---|
| Definition | cytochrome P450 family 26 subfamily A member 1 |
| Date | Results | Publications |
|---|---|---|
| 2020-08-15 19:03:00 | 26A1 modulates uterine dendritic cells in mice early pregnancy. | 31148354 |
| 2020-08-01 13:31:00 | This review highlights the current knowledge of structure-function of CYP26 enzymes and focuses on their role in human retinoid metabolism in different tissues. | 31419517 |
| 2020-03-28 10:27:00 | The combined effects of the novel CYP26 single nucleotide polymorphisms -environment approach may predict the risk of occurrence of oral malignant disorders. | 31465460 |
| 2019-02-23 11:47:00 | We applied whole-genome sequencing (WGS) on 9 trios where the probands are sporadically affected with the most severe form of the disorder and harbor no coding sequence variants affecting the function of known Hirschsprung disease (HSCR) genes. We found de novo protein-altering variants in three intolerant to change genes-CCT2, VASH1, and CYP26A1-for which a plausible link with the enteric nervous system (ENS) exists | 29483666 |
| 2017-10-07 12:11:00 | Accumulating evidence suggest that cytochrome P450 (CYP26), the primary retinoid-inactivating enzyme, plays a critical role in the integration of two neoplastic molecular programs: the retinoid metabolism and Hedgehog pathways. (Review) | 28754309 |
| Type | IDs |
|---|---|
| Synonymous | CP26, CYP26, P450RAI, P450RAI1 |
| Gene |
UniProtKB-ID:
CP26A_HUMAN
UniprotKB:
O43174
UniParc:
UPI000013E48F,
UPI000013C849
EMBL:
AK027560,
AF005418,
CH471066,
AL358613
Ensembl:
ENSG00000095596
KO:
hsa:1592
|
| Nucleutide sequences |
EMBL-CDS:
BAG51346.1,
EAW50079.1,
AAB88881.1,
EAW50078.1
Ensembl_TRS:
ENST00000224356,
ENST00000371531
|
| Protein sequencees |
Ensembl_PRO:
ENSP00000224356,
ENSP00000360586
RefSeq:
NP_476498.1,
NP_000774.2
|
| Others |
UniRef100:
UniRef100_O43174
UniRef90:
UniRef90_O43174
UniRef50:
UniRef50_P79739
UniGene:
Hs.150595
CCDS:
CCDS7427.1,
CCDS7426.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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