| Type | Description |
|---|---|
| Definition | PRKC, apoptosis, WT1, regulator |
| Date | Results | Publications |
|---|---|---|
| 2019-07-20 11:12:00 | Lack of Par-4 increased the expression of p21 and delayed chronic lymphocytic leukemia (CLL) growth in Emu-Tcl1 mice. | 29695515 |
| 2018-08-25 11:34:00 | Induced by endoplasmic reticulum stress Par-4 acts as an important defense mechanism against mycobacterial infection and regulates cancer. | 27552917 |
| 2017-07-01 10:36:00 | Findings reveal a novel role for Par-4/NF-kappaB in islet beta cell apoptosis and type 2 diabetes. | 27340675 |
| 2016-03-05 11:26:00 | Par4, CEBPB and FAK form a senescence signaling pathway, playing roles in modulating cell survival, growth, apoptosis, EMT and self-renewal | 25472717 |
| 2012-08-25 10:45:00 | a novel mechanism of apoptosis induction by PAR-4/ceramide-enriched exosomes, which may critically contribute to Alzheimer disease. | 22532571 |
| Type | IDs |
|---|---|
| Synonymous | 2310001G03Rik, PAR4, Par-4 |
| Gene |
UniProtKB-ID:
PAWR_MOUSE
UniprotKB:
Q925B0
UniParc:
UPI0000DA7CCA,
UPI00001C4849
EMBL:
DQ363525,
AF377871,
DQ449074,
DQ449073
Ensembl:
ENSMUSG00000035873
KO:
mmu:114774
|
| Nucleutide sequences |
EMBL-CDS:
ABC96645.1,
ABE27591.1,
AAK55414.1,
ABE27592.1
Ensembl_TRS:
ENSMUST00000218332,
ENSMUST00000095313
|
| Protein sequencees |
Ensembl_PRO:
ENSMUSP00000092951,
ENSMUSP00000151457
RefSeq:
NP_473397.1,
XP_036011407.1
|
| Others |
UniRef100:
UniRef100_Q925B0
UniRef90:
UniRef90_Q62627
UniRef50:
UniRef50_Q62627
UniGene:
Mm.391419
CCDS:
CCDS36053.1
|
| {{proteinIndex+1}} | mRNA | Protein | UniprotKB | Description | ||||
|---|---|---|---|---|---|---|---|---|
| Refseq |
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| Conserved domain | Region: {{conservedDomain.region == '' || conservedDomain.region == null ? "-": conservedDomain.region}} GFID: {{conservedDomain.gfid == '' || conservedDomain.gfid == null ? "-": conservedDomain.gfid}} Family: {{conservedDomain.family == '' || conservedDomain.family == null ? "-": conservedDomain.family}} CDD: {{conservedDomain.cdd}} - |
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