Annu Int Conf IEEE Eng Med Biol Soc. 2020 Jul;2020:2353-2356. doi:10.1109/EMBC44109.2020.9175466
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摘要
in atrial myocytes with the rs13143308T risk variant. Further computational analysis revealed that these calcium-mediated triggered activities were mainly linked to the gain of SERCA2a function but not the RyR2 dysfunction. Taken together, our model provides a powerful tool for assessing the impact of genetic variants in Pitx2, and these simulated results enhance our understanding of the molecular mechanisms underlying Pitx2-induced AF.
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基因功能
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原始数据
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文献解读
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