Cross-talk between CD38 and TTP Is Essential for Resolution of Inflammation during Microbial Sepsis.

Cell Rep. 2020 Jan 28;30(4):1063-1076.e5

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levels, suppresses the acute inflammatory response and decreases Rheb expression, inhibits mTORC1, and induces autophagolysosomes for bacterial clearance. TTP may represent a mechanistic target of anti-inflammatory agents, such as carbon monoxide. TTP mediates crosstalk between acute inflammation and autophagic clearance of bacteria from damaged tissue in the resolution of inflammation during sepsis.

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Cross-talk between CD38 and TTP Is Essential for Resolution of Inflammation during Microbial Sepsis.

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