Beyond ER: Regulating TOM-Complex-Mediated Import by Ubx2.
Trends Cell Biol.2019 Sep;29(9):687-689. Epub 2019 Jul 26
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摘要
Despite the progress in understanding the molecular responses to mitochondrial damage, responses to aberrant accumulation of mitochondrial precursor proteins and mitochondrial import defects remain poorly understood. Recent work (MÃ¥rtensson et al., Nature, 2019) has unveiled a pathway similar to endoplasmic-reticulum-associated degradation (ERAD) in fine-tuning the fidelity of translocase of the outer mitochondrial membrane (TOM) complex-mediated mitochondrial import.
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基因功能
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文献解读
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