Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease.
Exp. Neurol.2018 Jan;299(Pt A):26-41. Epub 2017 Sep 30
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摘要
mice mimic the neuropathological and clinical abnormalities of the classical early-onset Tay-Sachs patients, and provide a suitable model for the future pre-clinical testing of potential treatments for this condition.
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基因功能
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原始数据
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文献解读
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