Recombinant human elafin promotes alveologenesis in newborn mice exposed to chronic hyperoxia.
Int. J. Biochem. Cell Biol.2017 Nov;92:173-182. Epub 2017 Aug 10
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摘要
BACKGROUND/AIMS:-rich gas. The potential of these molecules to improve endothelial function and to ameliorate severe bronchopulmonary dysplasia (BPD) during lung development is not yet understood. We sought to determine whether the intratracheal treatment of newborn mice with the elastase inhibitor elafin would prevent hyperoxia-induced lung elastin degradation and the cascade of events that cause abnormal alveologenesis. METHODS:. RESULTS:exposure. CONCLUSIONS:-induced lung injury in neonates.
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基因功能
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原始数据
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文献解读
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