1.5 promoted by the Î²2 subunit is defective due to a Î²2 mutation associated with Brugada syndrome.

Biol. Cell. 2017 Jul;109(7):273-291. doi:10.1111/boc.201600085

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BACKGROUND INFORMATION: 1.5, the Î± subunit of the major cardiac voltage-gated sodium channel. This channel forms a protein complex including one or two associated Î² subunits as well as other proteins. RESULTS: 1.5 surface localisation. CONCLUSIONS: 1.5, which can be affected due to Î²2 mutations associated with channelopathies. SIGNIFICANCE: 1.5 trafficking and localisation, which must influence cell excitability and electrical coupling in the heart. This study will contribute to knowledge on development of arrhythmias.

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1.5 promoted by the Î²2 subunit is defective due to a Î²2 mutation associated with Brugada syndrome.

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