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The AtrbohF-dependent regulation of ROS signaling is required for melatonin-induced salinity tolerance in Arabidopsis.

Free Radic. Biol. Med.2017 Jul;108:465-477. Epub 2017 Apr 12
Ziping Chen 1 , Yanjie Xie 1 , Quan Gu 1 , Gan Zhao 1 , Yihua Zhang 1 , Weiti Cui 1 , Sheng Xu 2 , Ren Wang 2 , Wenbiao Shen 3
Ziping Chen 1 , Yanjie Xie 1 , Quan Gu 1 , Gan Zhao 1 , Yihua Zhang 1 , Weiti Cui 1 , Sheng Xu 2 , Ren Wang 2 , Wenbiao Shen 3
+ et al

[No authors listed]

Author information
  • 1 College of Life Sciences, Laboratory Center of Life Sciences, Nanjing Agricultural University, Nanjing, Jiangsu, China.
  • 2 Institute of Botany, Jiangsu Province and Chinese Academy of Science, Nanjing, Jiangsu, China.
  • 3 College of Life Sciences, Laboratory Center of Life Sciences, Nanjing Agricultural University, Nanjing, Jiangsu, China. Electronic address: wbshenh@njau.edu.cn.

摘要

efflux might be mediated by AtrbohF-dependent The addition of melatonin could intensify the increased antioxidant defence in stressed wild-type but not in atrbohF mutant, both of which were confirmed by the histochemical staining for production and lipid peroxidation during the later period of stress. Collectively, our genetic and molecular evidence revealed that the AtrbohF-dependent duanyu1670 signaling is required for melatonin-induced salinity tolerance via the reestablishment of ion and redox homeostasis.

KEYWORDS: Arabidopsis, AtrbohF, Ion homeostasis, Melatonin, Reactive oxygen species, Redox homeostasis

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