[No authors listed]
efflux might be mediated by AtrbohF-dependent The addition of melatonin could intensify the increased antioxidant defence in stressed wild-type but not in atrbohF mutant, both of which were confirmed by the histochemical staining for production and lipid peroxidation during the later period of stress. Collectively, our genetic and molecular evidence revealed that the AtrbohF-dependent duanyu1670 signaling is required for melatonin-induced salinity tolerance via the reestablishment of ion and redox homeostasis.
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