Deficiency of a sulfotransferase for sialic acid-modified glycans mitigates Alzheimer's pathology.
Proc. Natl. Acad. Sci. U.S.A.2017 Apr 04;114(14):E2947-E2954. Epub 2017 Mar 20
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摘要
mice manifested reduced cerebral amyloid-β deposition. GlcNAc6ST1-synthesizing sialylated KS thus modulates AD pathology. Inhibition of KS synthesis by targeting GlcNAc6ST1 may therefore be beneficial for controlling AD pathogenesis.
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基因功能
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原始数据
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文献解读
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