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Genetics. 2017 May;206(1):481-496. Epub 2017 Mar 14
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摘要
mutant defective in endonucleolytic activity, the same mutant lacking Ctp1, or the triple mutant also lacking the putative hairpin nuclease Pso2 showed wild-type levels of repair. Thus, MRN may act to recruit the hairpin opening activity that allows subsequent repair.
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基因功能
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原始数据
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文献解读
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