Inhibition of infarction-induced sympathetic innervation with endothelin receptor antagonism via a PI3K/GSK-3β-dependent pathway.
Lab. Invest.2017 Mar;97(3):243-255. Epub 2016 Dec 19
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摘要
receptor antagonism is a mediator to attenuate sympathetic hyperinnervation probably through restoration of signaling pathway, a potential pharmacological target for arrhythmias after infarction.
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原始数据
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文献解读
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