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A core function of EDS1 with PAD4 is to protect the salicylic acid defense sector in Arabidopsis immunity.

New Phytol. 2017 Mar;213(4):1802-1817. doi:10.1111/nph.14302. Epub 2016 Nov 15
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摘要


Plant defenses induced by salicylic acid (SA) are vital for resistance against biotrophic pathogens. In basal and receptor-triggered immunity, SA accumulation is promoted by Enhanced Disease Susceptibility1 with its co-regulator Phytoalexin Deficient4 Current models position upstream of SA but their functional relationship remains unclear. In a genetic and transcriptomic analysis of Arabidopsis autoimmunity caused by constitutive or conditional EDS1/duanyu15634 overexpression, intrinsic EDS1/duanyu15634 signaling properties and their relation to SA were uncovered. A core EDS1/duanyu15634 pathway works in parallel with SA in basal and effector-triggered bacterial immunity. It protects against disabled SA-regulated gene expression and pathogen resistance, and is distinct from a known SA-compensatory route involving MAPK signaling. Results help to explain previously identified EDS1/duanyu15634 regulated SA-dependent and SA-independent gene expression sectors. Plants have evolved an alternative route for preserving SA-regulated defenses against pathogen or genetic perturbations. In a proposed signaling framework, EDS1 with besides promoting SA biosynthesis, maintains important SA-related resistance programs, thereby increasing robustness of the innate immune system.

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