, causes defects in plasminogen binding and inflammatory macrophage recruitment in vivo.
J. Thromb. Haemost.2017 Jan;15(1):155-162. doi:10.1111/jth.13532. Epub 2016 Nov 28
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摘要
deficiency is not compatible with survival of the species. SUMMARY:females. This new mouse model will be important for future studies aimed at delineating the role of cell surface plasminogen activation in challenge and disease models in vivo.
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基因功能
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原始数据
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文献解读
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