A novel cell death gene acts to repair patterning defects in Drosophila melanogaster.

Genetics. 2014 Jun;197(2):739-42. Epub 2014 Mar 25

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摘要

Cell death is a mechanism utilized by organisms to eliminate excess cells during development. Here, we describe a novel regulator of caspase-independent cell death, Mabiki (Mabi), that is involved in the repair of the head patterning defects caused by extra copies of bicoid in Drosophila melanogaster. Mabiki functions together with caspase-dependent cell death mechanisms to provide robustness during development.

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A novel cell death gene acts to repair patterning defects in Drosophila melanogaster.

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